幽门螺杆菌感染引发非酒精性脂肪性肝病机制的研究进展  

Mechanisms of non-alcoholic fatty liver disease induced by Helicobacter pylori

作　　者：袁晨[1] 芦琳琳[2] 辛永宁[1] 吴梦娟[1] 宣世英[1] 

机构地区：[1]南京医科大学青岛临床医学院消化科,山东青岛266011 [2]青岛市市立医院中心实验室,山东青岛266011

出　　处：《中国肝脏病杂志（电子版）》2016年第3期26-29,共4页Chinese Journal of Liver Diseases：Electronic Version

基　　金：青岛市民生科技计划(No.14-2-3-17-nsh)

摘　　要：非酒精性脂肪性肝病(NAFLD)是我国突出的公众健康问题,由其导致的经济负担不断加重,已成为当下的研究热点,但其致病机制尚不明确。研究表明,幽门螺杆菌感染是NAFLD发病的独立危险因素之一。本文就幽门螺杆菌引发NAFLD作用机制的最新研究进展进行综述,以期为NAFLD的治疗带来新的曙光。Non-alcoholic fatty liver disease（NAFLD） is currently considered to be a serious problem to public health in our country. The mechanism of NAFLD remains unclear and has been a topic at present. Accumulating evidences have implicated Helicobacter pylori infection as an independent risk factor for the development of NAFLD. This review summarizes the evolution of the pathogenetic role of Helicobacter pylori in NAFLD. Thus, eradication of Helicobacter pylori may provide new treatment strategies to halt the progression of NAFLD.

关 键 词：脂肪肝 非酒精性 幽门螺杆菌 感染 

分 类 号：R575.5[医药卫生—消化系统]