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机构地区:[1]湖南农业大学植物保护学院,湖南长沙410128
出 处:《大豆科学》2017年第3期475-479,共5页Soybean Science
基 金:湖南农业大学青年科学基金(14QN19);湖南农业大学"神农学者"计划(2013);湖南省教育厅项目(62021000032)
摘 要:大豆胞囊线虫病(Heterodera glycines,soybean cyst nematode,SCN)是大豆生产上的重要病害,其特点为危害重、分布广、难防治,每年对大豆生产造成极大的损失。种植大豆抗性新品种是防治SCN目前最为有效的措施,研究大豆对SCN侵染的应答机制,是培育大豆持久抗病品种的前提,对加快抗线虫品种选育及SCN的防控具有重要的意义。本文综述了大豆对SCN侵染的组织细胞学应答机制;介绍了大豆在SCN侵染后酶系变化及酚类代谢的生理生化应答机制;从分子水平阐明了SCN侵染后大豆的基因转录变化,差异蛋白及DNA甲基化的应答机制,以期为大豆胞囊线虫病害的进一步研究与防治提供参考。Soybean cyst nematode (Heterodera glycines, SCN) is an important disease on soybean production, which is char- acterized by heavy damage, widespread and difficult to control, and causes great losses every year. Planting of resistant soy- bean varieties is the most effective measure to control SCN. Research of soybean response mechanism to SCN is the prerequi- site for cultivating durable resistant varieties, and has great significance to speed up the anti-nematode breeding and the con- trol of SCN. Here we reviewed the histocytology mechanism of soybean against SCN, soybean physiological and biochemical mechanisms of enzyme changes and phenolic metabolism after SCN infection. And also the mechanism of soybean gene tran- scription variation , protein difference and DNA methylation after SCN infection was introduced at the molecular level. Hopely, it will be useful information for further study of SCN and its control.
关 键 词:大豆胞囊线虫(SCN) 组织细胞学 酶系 转录水平 DNA甲基化
分 类 号:S435.651[农业科学—农业昆虫与害虫防治]
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