卒中后认知功能障碍发病机制的研究进展  

The pathogenesis of post-stroke cognitive impairment

作　　者：许志祥[1] 吴云成[1] Xu Zhixiang;Wu Yuncheng(Department of Neurology,Shanghai General Hospital,School of Medicine,Shanghai Jiao Tong University,Shanghai 200080,China)

机构地区：[1]上海交通大学医学院附属第一人民医院神经内科,上海200080

出　　处：《上海医药》2018年第17期5-8,17,共5页Shanghai Medical & Pharmaceutical Journal

摘　　要：卒中后认知功能障碍(post-stroke cognitive impairment,PSCI)是指在卒中这一临床事件发生后6个月内出现的达到认知功能障碍诊断标准的一种综合征,对其进行早期诊断及干预具有重要的临床意义。了解PSCI的发病机制对干预和治疗PSCI具有重要的指导作用。本文概要介绍有关PSCI发病机制的研究进展。Post-stroke cognitive impairment(PSCI)generally refers to a syndrome that meet the diagnostic criteria of cognitive impairment within 6 months after stroke.Early diagnosis and intervention are of great significance and understanding its pathogenesis plays an important role in the intervention treatment.We summarize the progress in the pathogenesis of PSCI in this review.

关 键 词：卒中后认知功能障碍 血管性痴呆症 发病机制 

分 类 号：R749.13[医药卫生—神经病学与精神病学]