缺血性肝炎临床特点分析  

Clinical analysis of 17 cases of ischemic hepatitis

作　　者：周万 Zhou Wan(University Town Hospital of Chongqing Medical University,Chongqing 401331,China)

机构地区：[1]重庆医科大学附属大学城医院,重庆401331

出　　处：《医药前沿》2019年第1期56-56,共1页Journal of Frontiers of Medicine

摘　　要：目的:讨论缺氧性肝炎的生化、临床特点,提高临床诊断认识.方法:对17例缺血性肝炎患者的资料数据进行分析.结果:缺血性肝炎患者血清血清丙氨酸转氨酶(ALT)、天门冬氨酸氨基转移酶(AST)、乳酸脱氢酶(LDH)测值明显升高,总胆红素(TBIL)水平轻度升高.缺血性肝炎的诱因主要为心力衰竭、感染性休克、低血容量性休克后等.结论:缺血性肝炎患者生化改变多以ALT、AST、LDH为主,呈显著升高趋势,病因纠正后可快速下降.缺血性肝炎死亡率较高,应提高临床认识,及早诊断与治疗.经积极治疗,存活患者的肝功能常能快速恢复.Objective To discuss the biochemical and clinical features of hypoxic hepatitis and to improve the clinical diagnosis.Methods The data of 17 patients with ischemic hepatitis were analyzed.Results Serum alanine aminotransferase(ALT),aspartate aminotransferase(AST),)lactate dehydrogenase(LDH)and total bilirubin(TBIL)were significantly increased in patients with ischemic hepatitis.The main causes of ischemic hepatitis are heart failure,septic shock,hypovolemic shock and so on.Conclusion ALXASTLDH is the main biochemical change in patients with ischemic hepatitis.Potential,cause of correction after the rapid decline.The mortality rate of ischemic hepatitis is high,so we should improve clinical understanding,diagnosis and treatment as soon as possible.After active treatment,the liver function of surviving patients can often recover quickly.

关 键 词：缺血性肝炎 乳酸脱氢酶 转氨酶 临床分析 

分 类 号：R575.1[医药卫生—消化系统]