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作 者:Andrea Tagliani
机构地区:[1]Department of Biosciences,Universitàdegli Studi di Milano,20133 Milan,Italy
出 处:《Molecular Plant》2020年第10期1349-1349,共1页分子植物(英文版)
摘 要:Stomatal closure upon pathogen attack is a process involving Ca2+entry into guard cells after perception of pathogen-associated molecular patterns(PAMPs),small microbial molecules recognized by plant receptors.A central role in this process is played by the signal transducing kinase BIK1(Monaghan et al.,2015),which regulates cytosolic Ca2+entry and reactive oxygen species production.However,the Ca2+channel responsible for pathogen-induced cytosolic Ca2+increase in guard cells remains unknown.Recently,in a Nature paper,Thor et al.(2020)reported the characterization of an Arabidopsis Ca2+-permeable channel,OSCA1.3,which is regulated by the BIK1 kinase to participate in this adaptive response(Figure 1).
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