Pgant4 and Tango1 Mediate Anoxia and Reoxygenation Injury  

作　　者：Qingqing Du Nastasia KHLim Yiling Xia Wangchao Xu Qichao Zhang Liyao Zhang Fude Huang Wenan Wang 

机构地区：[1]Department of Neurology,Xinhua Hospital Affiliated to Shanghai Jiaotong University School of Medicine,Shanghai 202150,China [2]Department of Neurology,Xinhua Hospital Chongming Branch Affiliated to Shanghai Jiaotong University School of Medicine,Shanghai 202150,China [3]Shanghai Institute of Materia Medica,University of the Chinese Academy of Sciences,Shanghai 201203,China [4]Shanghai Advanced Research Institute,University of the Chinese Academy of Sciences,Shanghai 201210,China [5]Nuo-beta Pharmaceutical Technology(Shanghai)Co.Ltd,Shanghai 201210,China [6]Institute of Neuroscience,University of the Chinese Academy of Sciences,Shanghai 200031,China [7]Sino-Danish College,University of the Chinese Academy of Sciences,Beijing 100049,China

出　　处：《Neuroscience Bulletin》2020年第12期1552-1557,共6页神经科学通报（英文版）

基　　金：the National Natural Science Foundation of China(81771416,81650110527 and 8197100);Shanghai Municipal Commission of Health and Family Planning(201740153);Key Discipline of Chongming District,Shanghai,China,2018。

摘　　要：Dear Editor,Diseases caused by ischemia,including coronary artery disease and stroke,are a leading cause of death worldwide[1].Restoration of the blood and oxygen supply after restriction also causes tissue damage by activating a number of pathological pathways,such as inflammatory,oxidative stress,and cell death pathways that are mediated by microRNAs and hypoxia-inducible factors[1,2].

关 键 词：DEATH damage ISCHEMIA 

分 类 号：R743.3[医药卫生—神经病学与精神病学]