Stressed axons craving for glial sugar: links to regeneration?  

作　　者：Elisabetta Babetto Bogdan Beirowski 

机构地区：[1]Hunter James Kelly Research Institute,Jacobs School of Medicine and Biomedical Sciences,University at Buffalo,Buffalo,NY,USA [2]Department of Biochemistry,Jacobs School of Medicine and Biomedical Sciences,University at Buffalo,Buffalo,NY,USA [3]Department of Pharmacology and Toxicology,Jacobs School of Medicine and Biomedical Sciences,University at Buffalo,Buffalo,NY,USA

出　　处：《Neural Regeneration Research》2022年第2期304-306,共3页中国神经再生研究（英文版）

基　　金：supported by grants from the National Institute of Health(R01NS111024);the Muscular Dystrophy Association(577844);Funding provided through the Empire State Development Corporation for Hunter James Kelly Research Institute grant nos.W753 and U446;the Hunter’s Hope Foundation(to BB)。

摘　　要：Extract The contrary but interrelated processes of axon degeneration and regeneration are the yin and yang of many neurodegenerative conditions. Here we discuss recent evidence for metabolic cross-talk between glia and injured axons regulating these processes. We especially focus on potential bioenergetic mechanisms as to how axon-flanking glia may promote regeneration.

关 键 词：DEGENERATION conditions. processes. 

分 类 号：Q421[生物学—神经生物学]