A neuron's ambrosia: non-autonomous unfolded protein response of the endoplasmic reticulum promotes lifespan  

作　　者：Stefan Homentcovschi Ryo Higuchi-Sanabria 

机构地区：[1]Department of Molecular&Cellular Biology,University of California,Berkeley,CA,USA

出　　处：《Neural Regeneration Research》2022年第2期309-310,共2页中国神经再生研究（英文版）

基　　金：funding support provided by the National Institute of Aging through grant 1K99AG065200-01A1 to RHS。

摘　　要：The ability to protect cellular components in the face of deleterious conditions, such as exposure to chemical poisons, damaging radiation, or excessive heat, is crucial to organismal viability.

关 键 词：autonomous DELETE exposure 

分 类 号：Q421[生物学—神经生物学]