原发性三叉神经痛发病机制研究年鉴(2021.7-2022.6)  

Progress of pathogenesis in primary trigeminal neuralgia:a yearbook of July 2021 to June 2022

作　　者：刘亚明[1] Liu Yaming(Department of Painology,The Third Hospital of Hebei Medical University,Shijiazhuang City,Hebei Province 050051,China)

机构地区：[1]河北医科大学第三医院疼痛科,石家庄市050051

出　　处：《中华疼痛学杂志》2022年第6期I0013-I0015,共3页Chinese Journal Of Painology

摘　　要：过去一年对三叉神经痛发病机制的研究集中在各种膜受体,如ATP门控P2X3受体、辣椒辣素受体等,为创新药物的研发指明新方向。对三叉神经痛患者研究中发现海马体结构的异常,为三叉神经痛引起的中枢改变提供了有力证据。In the past year,researches on the pathogenesis of trigeminal neuralgia focused on a variety of membrane receptors,such as ATP-gated P2X3 receptor and capsaicin receptor,and these indicate the new directions for the development of innovative drugs.Patients with trigeminal neuralgia have abnormal structures in the hippocampus,which provides strong evidence for the central changes caused by trigeminal neuralgia.

关 键 词：三叉神经痛 发病机制 膜受体 海马结构 年鉴 

分 类 号：R745.11[医药卫生—神经病学与精神病学]