提高对睫状后动脉阻塞及脉络膜缺血的认识(下)  

Improving the recognition of posterior ciliary artery occlusion and choroidal ischemia(II)

作　　者：黄厚斌[1,2] Huang Houbin(Senior Department of Ophthalmology,PLA General Hospital,Beijing 100853,China;Department of Ophthalmology,Hainan Hospital of PLA General Hospital,Sanya Hainan,572013,China)

机构地区：[1]解放军总医院眼科医学部,北京100853 [2]解放军总医院海南医院眼科,海南三亚572013

出　　处：《眼科》2024年第3期169-177,共9页Ophthalmology in China

基　　金：海南省重点研发计划高新技术项目(ZDYF2024GXJS032);军队后勤科研项目保健专项课题(23BJZ37);海南省卫生健康行业科研项目(22A200351)。

摘　　要：睫状后动脉阻塞(PCAO)可有不同的表现型式,在疾病的不同时期,其临床表现也大为迥异,从超急性期的眼底无任何异常,至晚期出现严重的色素紊乱,而且缺血坏死的形态、大小、范围、分布等均差异很大。由于复杂的代偿机制,同一支PCAO的区域内眼底的不同部位还可以表现为不同程度的缺血。PCAO造成的视盘的缺血因动脉炎性、非动脉炎性、糖尿病性等,其发病机制和临床表现也有很大的差异。The posterior ciliary artery occlusion(PCAO)can manifest multiple patterns,with totally different clinical characteristics associated with stages,ranging from unrecognizable retinal abnormality to severe pigmented fundus.Great variability can be noted in the distributions,morphology,sizes and extents of ischemia lesions.What’s more,different degrees of ischemia can be appeared in a same fundus of PCAO because of complex compensatory mechanisms.The clinical features of optic nerve head ischemia caused by PCAO also varies depending on different etiologies,such as non-arteritic,arteritic,diabetic.

关 键 词：睫状后动脉 睫状后动脉阻塞 脉络膜缺血 视神经缺血 

分 类 号：R773[医药卫生—眼科]