颞下颌关节骨关节炎分子致病机制的研究进展  

Research Progress on the Molecular Pathogenesis of Temporomandibular Joint Osteoarthritis

作　　者：陈一晗 张善勇[2] 丁昱 张莉[1] CHEN Yihan;ZHANG Shanyong;DING Yu;ZHANG Li(Stomatological Center,Affiliated Hospital of Yunnan University,Kunming Yunnan 650021;Shanghai Ninth People’s Hospital,Shanghai Jiao Tong University School of Medicine,Shanghai 200011,China)

机构地区：[1]云南大学附属医院口腔医学中心,云南昆明650021 [2]上海交通大学医学院附属第九人民医院口腔外科,上海200011

出　　处：《昆明医科大学学报》2024年第7期1-5,共5页Journal of Kunming Medical University

基　　金：国家自然科学基金资助项目(82071135);云南省省级临床重点专科开放课题(ZKF2024033)。

摘　　要：颞下颌关节骨关节炎(temporomandibular joint osteoarthritis,TMJOA)是发生于颞颌关节结构的1种慢性退行性病变,但TMJOA的发病机制仍未完全阐明。随着研究方法的进步,TMJOA的研究开始深入到基因层面,对近年来颞下颌关节骨关节炎发病机制的分子生物研究的进展进行综述。Temporomandibular joint osteoarthritis(TMJOA)is a chronic degenerative condition affecting the structures of the temporomandibular joint.Despite ongoing research efforts,the pathogenesis of TMJOA is not yet fully understood.Recent advancements in research techniques have allowed for a deeper exploration of the genetic factors underlying TMJOA.This article will provide an overview of the most recent developments in molecular biology research related to the pathogenesis of temporomandibular joint osteoarthritis.

关 键 词：颞下颌关节 骨关节炎 致病机制 

分 类 号：R578[医药卫生—消化系统]