TMEM63B regulates postnatal development of cochlear sensory epithelia via thyroid hormone signaling  

作　　者：Chang Ye Tian-Zi Zhang Yan-Yu Zang Yun Stone Shi Guoqiang Wan 

机构地区：[1]State Key Laboratory of Pharmaceutical Biotechnology and MOE Key Laboratory of Model Animal for Disease Study,Model Animal Research Center,Medical School,Nanjing University,Nanjing,Jiangsu 210032,China [2]Guangdong Institute of Intelligence Science and Technology,Zhuhai,Guangdong 519031,China

出　　处：《Journal of Genetics and Genomics》2024年第6期673-676,共4页遗传学报（英文版）

基　　金：supported by the National Natural Science Foundation of China(82171136 and 81970888 to G.W.,32330044 to Y.S.S.);Fundamental Research Funds for the Central Universities(021414380533toG.W.).

摘　　要：Sound transmission occurs in the cochlea,a complex and ingenious subdivision in the inner ear.The structure of the cochlea develops structurally and functionally by the time before two postnatal weeks(the time of hearing onset)in mice(Geal-Dor et al.,1993).Greater epithelial ridge(GER,also known as Kolliker's organ)is a transient cochlear structure containing a group of columnar epithelial supporting cells surrounding the inner hair cells(IHCs).

关 键 词：COCHLEAR al. SUPPORTING 

分 类 号：Q965[生物学—昆虫学]