急性缺血性卒中并发应激性高血糖的研究进展  

Research Progress in Acute Ischemic Stroke Complicated with Stress Hyperglycemia

作　　者：赵智艳 徐云瑀 尚正福 张颖[1] 缪薇[1] 

机构地区：[1]昆明医科大学第二附属医院神经内科,云南 昆明 [2]保山市第二人民医院神经内科,云南 保山

出　　处：《临床医学进展》2024年第5期1700-1707,共8页Advances in Clinical Medicine

摘　　要：应激性高血糖在急性缺血性卒中病人中较为常见,应激性高血糖会增加急性缺血性卒中患者出现出血转化、神经功能恶化、功能恢复不良,甚至死亡的风险,但应激性高血糖影响急性缺血性脑卒中患者预后的机制尚未明确。本文将围绕急性缺血性卒中后应激性高血糖的成因、应激性高血糖对急性缺血性卒中的损伤及保护作用、应激性高血糖的水平与急性缺血性卒中患者预后的相关性做一综述,旨在为临床治疗提供理论依据。Stress hyperglycemia is relatively common in patients with acute ischemic stroke. Stress hyperglycemia may increase the risk of hemorrhage transformation, neurological deterioration, poor functional recovery and even death in patients with acute ischemic stroke. However, the pathophysiological mechanism of stress hyperglycemia’s effect on acute ischemic stroke remains unclear. In this paper, we will review the causes of stress hyperglycemia after acute ischemic stroke, the damage and protective effect of stress hyperglycemia on acute ischemic stroke, and the correlation between the level of stress hyperglycemia and the prognosis of patients with acute ischemic stroke, aiming to provide theoretical basis for clinical treatment.

关 键 词：应激性高血糖 急性缺血性卒中 机制 预后 

分 类 号：R74[医药卫生—神经病学与精神病学]