镉对草鱼的急性毒性效应及SOD的影响  

SOD Effect of Cadmium Chloride on Acute Toxicity of Grass Carp

作　　者：王桂燕[1] 胡筱敏[1] 周启星[2] 孙学奇[3] 

机构地区：[1]东北大学资源与土木工程学院,辽宁沈阳110004 [2]中国科学院沈阳应用生态研究所陆地生态过程重点实验室,辽宁沈阳110016 [3]沈阳市消防局,辽宁沈阳110013

出　　处：《东北大学学报（自然科学版）》2007年第12期1758-1761,共4页Journal of Northeastern University(Natural Science)

基　　金：国家重点基础研究发展规划项目(2004CB418503);国家自然科学基金重点项目(20337010)

摘　　要：以静水生物测试法研究了氯化镉对草鱼的急性毒性效应及其肝胰脏、肾脏和鳃组织的超氧化物歧化酶(SOD)的影响.结果表明:镉对草鱼的急性毒性作用时间为24,48,72和96 h的半数致死质量浓度(LC50)分别是45.58,34.81,28.63和24.05 mg.L-1,因此镉对草鱼的生态毒性为高毒.Cd2+低浓度时,肝胰脏和肾脏组织SOD活性均为"先降后升再降",而Cd2+为高浓度时SOD活性始终处于被显著抑制(p<0.05);鳃组织在低Cd2+污染时SOD活性为"先降后升",而高浓度的Cd2+污染时其SOD活性为"先降后升再降".但是草鱼鳃组织的SOD活性明显低于肝胰脏和肾脏的SOD活性.The acute toxicity of cadmium chloride to grass carp was investigated by stagnophile testing, as well as the effect of superoxide dismutase systems （SOD） on grass carp＇s liver, kidney and gill tissues. The results showed that the LC50 for grass carp are 45.58, 34.81, 28.63 and 24.05 mg·L^-1 when the acting time of acute toxicity of cadmium chloride are 24, 48, 72 and 96 h, respectively. The high ecological toxicity of cadmium is thus proved to grass carp. The activity of SOD in grass carp＇s liver and kidney tissues decreases first then increases and decreases once again when Cd^2＋ presents low concentration pollution, while the activity of SOD is inhibited obvious all along （p 〈 0.05） when Cd^2＋ presents high concentration pollution. However, the activity of SOD in grass carp＇s gill tissues decreases first then increases when Cd^2＋ presents low concentration pollution, while the activity decreases first then increases and then decreases once again when Cd^2＋ presents high concentration pollution. It was revealed that the activity of SOD in grass carp＇s gill tissues is much lower than in liver and kidney tissues.

关 键 词：氯化镉 毒性效应 草鱼(Ctenopharyngodon idellus) 半数致死质量浓度(LC50) 超氧化物歧化酶 (SOD) 

分 类 号：X171.5[环境科学与工程—环境科学]