Fine-tuning ROS homeostasis by ROD1 is a battleground between rice and Magnaporthe oryzae  

作　　者：Yoji Kawano 

机构地区：[1]CAS Center for Excellence in Molecular Plant Sciences,Shanghai Center for Plant Stress Biology,Chinese Academy of Sciences,Shanghai 201602,China [2]Kihara Institute for Biological Research,Yokohama City University,Yokohama,Kanagawa 244-0813,Japan [3]Institute of Plant Science and Resources,Okayama University,2-20-1,Chuo,Kurashiki,Okayama 710-0046,Japan

出　　处：《Molecular Plant》2021年第12期1979-1981,共3页分子植物（英文版）

基　　金：supported by JSPS KAKENHI(20H02988,21H05035);the Ohara Foundation;the Yakumo Foundation for Environmental Science;Ryobi Teien Memory Foundation;the Joint Usage/Research Center,Institute of Plant Science and Resources.

摘　　要：When plants recognize pathogens,reactive oxygen species(ROS)homeostasis is optimized to elicit immune responses.Pathogens also manipulate ROS homeostasis in host plants,which increases susceptibility to pathogens.A recent study found a novel regulation mechanism of ROS homeostasis through a rice susceptibility gene(ROD1)(Gao et al.,2021).The loss of function of ROD1 confers high resistance against three major rice pathogens but decreases yield,and an allele of ROD1 improves basal disease resistance without reducing yield.The current study sheds light on the fine-tuning of ROS ho-meostasis by ROD1 in rice and on how the Magnaporthe oryzae effector Avr-Piz-t highjacks on ROS homeostasis in rice.

关 键 词：HOMEOSTASIS MAGNAPORTHE SUSCEPTIBILITY 

分 类 号：S435.111.41[农业科学—农业昆虫与害虫防治]