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作 者:Yiran Yang Yan Li Yingjie Xu Hanxiao Zhang Yuhan Diao Shihao Chen Ye Cui Chris J.Corrigan Wei Wang Sun Ying
机构地区:[1]Department of Immunology,School of Basic Medical Sciences,Capital Medical University,Beijing,China [2]Department of Otorhinolaryngology Head and Neck Surgery,Beijing Tongren Hospital,Capital Medical University,Beijing Institute of Otorhinolaryngology,Key Laboratory of Otorhinolaryngology Head and Neck Surgery,Ministry of Education,Beijing Key Laboratory of Nasal Diseases,Beijing,100730,China [3]School of Rehabilitation,Capital Medical University,Beijing Bo’ai Hospital,China Rehabilitation Research Center,Beijing,China [4]Division of Asthma,Allergy&Lung Biology,MRC&Asthma UK Centre in Allergic Mechanisms of Asthma,King’s College London,London,UK
出 处:《Cellular & Molecular Immunology》2021年第11期2569-2571,共3页中国免疫学杂志(英文版)
基 金:This work was supported by grants from the National Natural Science Foundation of China(81471594,81700026,81971510,and 82071805).
摘 要:Group 2 innate lymphoid cells(ILC2s)are now known to play prominent roles in many inflammatory responses involving the production of Th2-type cytokines,which contribute to parasitic infections,tissue repair,and metabolic homeostasis[1].More recently,it has emerged that ILC2s may act as antigen-presenting cells,although corresponding evidence of phagocytic activity of these cells is currently scarce[2,3].Herein,we investigated whether ILC2s can phagocytose bacteria,through in vitro experiments.
关 键 词:LYMPHOID TH2 HOMEOSTASIS
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