Actin(g) toward a revised understanding of the role of cytoskeletal dynamics in neuronal bioenergetics  

作　　者：Sabrina M.Holland Gianluca Gallo 

机构地区：[1]Department of Neural Sciences,Shriners Pediatric Research Center,Lewis Katz School of Medicine at Temple University,Philadelphia,PA,USA

出　　处：《Neural Regeneration Research》2024年第10期2109-2110,共2页中国神经再生研究（英文版）

基　　金：supported by National Institute of Health awards NS118000 and NS128049 (to GG)。

摘　　要：Neurons are energy-demanding cells.Disruptions in energy metabolism can quickly interrupt neuronal function,leading to cell death and neurodegeneration.For instance,ischemia rapidly depletes adenosine triphosphate(ATP)thereby disrupting energy-dependent cellular processes crucial for homeostasis,and axon degeneration is preceded by a collapse of axonal ATP levels.

关 键 词：METABOLISM HOMEOSTASIS DEGENERATION 

分 类 号：Q421[生物学—神经生物学]