Brain scarring in infants:immunological insights from a neonatal hypoxic-ischemic encephalopathy model  

作　　者：Pedro Moreno Pimentel-Coelho 

机构地区：[1]Carlos Chagas Filho Biophysics Institute,Federal University of Rio de Janeiro,Rio de Janeiro,Brazil

出　　处：《Neural Regeneration Research》2025年第10期2909-2910,共2页中国神经再生研究（英文版）

基　　金：supported by Fundação de AmparoàPesquisa do Estado do Rio de Janeiro(FAPERJ,E-26/010.002160/2019,E-26/203.227/2017,E-260003/001177/2020,and E-26/201.279/2021);Conselho Nacional de Desenvolvimento Científico e Tecnológico(CNPq,313757/2020-8,311188/2023-0)(to PMPC).

摘　　要：Neonatal hypoxic-ischemic encephalopathy(HIE)is a significant cause of disability in children.Improving brain function and accelerating neurological recovery may require a combination of neuroprotective and pro-regenerative treatments at different stages of HIE.While the first hours after the neonatal insult are the most critical period for neuroprotection,the existence of secondary and tertiary mechanisms of brain injury offers the possibility of preventing delayed neurodegeneration in the subsequent days,weeks,or months(Levison et al.,2022).

关 键 词：NEONATAL protective HYPOXIC 

分 类 号：R742[医药卫生—神经病学与精神病学]