The authors gratefully acknowledge the financial support from the National Natural Science Foundation of China (81070220, 81170278, and 81100213); the Heng Yang Joint Funds of Hunan Provincial Natural Sciences Foundation of China (10JJ9019); the Hunan Provincial Natural Sciences Foundation of China (06jj5058); the Science & Technology Department Funds of Heng Yang of Hunan Province (2010kj17 and 2010kj41); and the Hunan Provincial Postgraduate Innovation Fund (CX2010B379).
Aim: To investigate the effects of the major component of high-density lipoprotein apolipoprotein A-I (apoA-I) on the development of atherosclerosis in LPS-challenged ApoE / mice and the underlying mechanisms. Meth...