This work was supported by the National Natural Science Foundation of China (No. 30200342, 30672466);the National Basic Research Development Program of China (No. 2006CB503806);the Special Fund for Promotion of Education, Ministry of Education of China.
为了验证心脏腺苷酸活化蛋白激酶(AMP-activated protein kinase,AMPK)是否为肾上腺素受体(adrenergic receptor,AR)的下游信号分子,本实验在大鼠心室肌源细胞和大鼠心脏中观察了α-AR对AMPK的激活作用,利用Western blot检测了AMPK-α...
In order to find candidate proteins that are potentially associated with the phenotype in neonatal rat cardiomyocytes treated with phenylephrine(PE), the differential proteins expression pattern after α-adrenergic re...
Impact of air pollution on cardiovascular d is eases has been established. As the main source of city air pollution, particulat e matter has been demonstrated an independent correlation with incidence and mor tality o...
This work was supported by the National Basic Research Priorities Programme of People's Republic of China (No. G2000056906); and the National Natural Science Foundation of China (No.30070872).
This work was supported by the National Basic Research Priorities Programme of China (G2000056906); and the National Natural Science Foundation of China (30070872); We appreciate technical assistance from Professor Eugene Chen (Morehouse School of Medi
为了解心肌肥厚时基因表达谱的变化规律,本实验复制了三种大鼠心肌肥厚模型:肾上腹主动脉缩窄(suprarenal abdominal aortic stenosis,SRS)、动静脉瘘(arterial-vein fistula,AVF)和去甲肾上腺素持续静脉输注(jugular vein infusion of ...
This work was supported by the National Basic Research Priorities Programme of China (No. G2000056906);the National Natural Science Foundation of China (No. 30270540; 30171083).