Does MgSO_(4) protect the preterm brain?Dissecting its role in the pathophysiology of hypoxic ischemic encephalopathy  

作　　者：Robert Galinsky Laura Bennet Alistair J.Gunn 

机构地区：[1]The Ritchie Centre,Hudson Institute of Medical Research,Melbourne,VIC,Australia [2]Department of Obstetrics and Gynaecology,Monash University,Melbourne,VIC,Australia [3]Department of Physiology,The University of Auckland,Auckland,New Zealand

出　　处：《Neural Regeneration Research》2024年第9期1861-1862,共2页中国神经再生研究（英文版）

基　　金：This work was supported by Health Research Council of New Zealand(grants 17/601 and 22/559);the Auckland Medical Research Foundation,the Lottery Health Grants Board of New Zealand,the C.J.Martin Postdoctoral Fellowship and project grant from the National Health and Medical Research Council of Australia(APP1090890 and APP1164954);the Victorian Government’s Operational Infrastructure Support Program(to RG).

摘　　要：Mitigating preterm encephalopathy continues to be one of the greatest challenges in perinatal medicine.Preterm encephalopathy is associated with high mortality,serious morbidity,and significant socio-economic impacts on the individuals,their families,and public health sectors and welfare systems that last a lifetime.The cost of disability associated with preterm brain injury continues to rise.Prevention of this injury,and disability,would significantly reduce this socioeconomic burden.

关 键 词：ENCEPHALOPATHY mortality HYPOXIC 

分 类 号：R742[医药卫生—神经病学与精神病学]