Navigating Epstein–Barr virus autoimmunity:role of NK cells and T cells in multiple sclerosis  

作　　者：Chu Xie Cong Sun Mu-Sheng Zeng 

机构地区：[1]State Key Laboratory of Oncology in South China,Guangdong Provincial Clinical Research Center for Cancer,Guangdong Key Laboratory of Nasopharyngeal Carcinoma Diagnosis and Therapy,Sun Yat-sen University Cancer Center,Guangzhou,P.R.China

出　　处：《Signal Transduction and Targeted Therapy》2024年第3期833-835,共3页信号转导与靶向治疗（英文）

基　　金：supported by grants from the National Key Research and Development Program of China(2022YFC3400900);Postdoctoral Fellowship Program of CPSF(GZB20230886);Chinese Postdoctoral Science Foundation(2023M743998);Natural Science Foundation of China(82030046);the Program for Guangdong Introducing Innovative and Entrepreneurial Teams(2019BT02Y198);Guangdong Science and Technology Department(2020B1212030004).

摘　　要：In a recent study published in Cell,Vietzen and colleagues discovered the novel role of natural killer(NK)cells and EBVspecific CD8+T cell response in controlling EBV-inducing autoimmune response and collectively defined virus and host genetics factors associated with a significantly raised risk of multiple sclerosis.

关 键 词：EPSTEIN SCLEROSIS KILLER 

分 类 号：R744.51[医药卫生—神经病学与精神病学]