Transient receptor potential channels and calcium dysregulation: a pathogenic duo in Parkinson's disease  

作　　者：Iqira Saeed Linlin Ma 

机构地区：[1]Griffith Institute for Drug Discovery,Griffith University,Nathan,Brisbane,QLD,Australia [2]School of Environment and Science,Griffith University Nathan,Brisbane,QLD,Australia

出　　处：《Neural Regeneration Research》2025年第3期808-810,共3页中国神经再生研究（英文版）

摘　　要：Parkinson's disease(PD) has a complex and multifactorial pathophysiology. Various studies, conducted both in pre-clinical models and PD patients, have reported a link between the disruption of calcium(Ca^(2+)) homeostasis and the subsequent development of PD. Ca^(2+) regulation is crucial for neuronal survival, differentiation,exocytosis at synapses,gene transcription,and proliferation.

关 键 词：HOMEOSTASIS CLINICAL subsequent 

分 类 号：R742.5[医药卫生—神经病学与精神病学]