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作 者:Ruoqi Zhou Yuwei Wang Xinxin Liu Xia Yu Dajing Xia Yihua Wu Yu Shi
机构地区:[1]State Key Laboratory for Diagnosis and Treatment of Infectious Diseases,National Clinical Research Center for Infectious Diseases,Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases,The First Affiliated Hospital,Zhejiang University School of Medicine,Hangzhou,Zhejiang,China [2]Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women’s Hospital,Zhejiang University School of Medicine,Hangzhou,Zhejiang,China
出 处:《Journal of Clinical and Translational Hepatology》2024年第10期892-895,共4页临床与转化肝病杂志(英文版)
基 金:supported by the National Key Research and Development Program of China(No.2022YFC2304501,No.2021YFC2301800);the Fundamental Research Funds for the Central Universities(226-2023-00127,2021FZZX001-41);the Chinese National Natural Science Foundation(No.81870425);the Medical Health Science and Technology Project of Zhejiang ProvincialHealth Commission(No.2022490480).
摘 要:Metabolic dysfunction-associated steatotic liver disease(MASLD)is a growing cause of chronic liver diseases worldwide,with a global prevalence of 38%.1 Patients with MASLD are at risk of developing liver-related complications,such as cirrhosis,hepatocellular carcinoma,and extra-hepatic adverse events.1 In recent years,endocrine-disrupting chemicals(EDCs)have been linked to the development and progression of MASLD.
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