S-GLUTATHIONYLATION

作品数：4被引量：1H指数：1
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Glutaredoxin-1 alleviates acetaminophen-induced liver injury by decreasing its toxic metabolites被引量：1
《Journal of Pharmaceutical Analysis》2023年第12期1548-1561,共14页Ying Xu Yan Xia Qinhui Liu Xiandan Jing Qin Tang Jinhang Zhang Qingyi Jia Zijing Zhang Jiahui Li Jiahao Chen Yimin Xiong Yanping Li Jinhan He 
supported by the National Natural Science Foundation of China(Grant Nos.:82025007,81930020,and 82170874);China Postdoctoral Science Foundation(Grant No.:2022M710099).
Excessive N-acetyl-p-benzoquinone imine(NAPQI)formation is a starting event that triggers oxidative stress and subsequent hepatocyte necrosis in acetaminophen(APAP)overdose caused acute liver failure(ALF).S-glutathion...
关键词：Glutaredoxin-1 S-GLUTATHIONYLATION ACETAMINOPHEN Toxic metabolites Cyp3a11 
Carbonic Anhydrase III S-Glutathionylation Is Necessary for Anti-Oxidant Activity
《Yangtze Medicine》2018年第4期244-254,共11页P. Roy M. Ireland S. Roy J. Craft C. Bartholomew 
Carbonic anhydrase isozyme CA3 protects cells against oxidative stress. Ectopic expression of murine Ca3, but not Ca2, protects proto-oncogene Evi1 expressing Rat1 fibroblast cells (ca3low) against hydrogen peroxide (...
关键词：CA3 CAIII Carbonic ANHYDRASE III S-GLUTATHIONYLATION APOPTOSIS ANTI-OXIDANT 
Quantification of Global Protein Disulfides and Thiol-Protein Mixed Disulfides to Study the Protein Dethiolation Mechanisms
《American Journal of Analytical Chemistry》2013年第10期9-19,共11页Lucia Coppo Raffaella Priora Sonia Salzano Pietro Ghezzi Paolo Di Simplicio 
The redox state of cellular thiols is widely studied because it was recently linked to many different diseases and pathologies. In this work we quantified the concentrations of protein disulfides (PSSP) and thiol-prot...
关键词：PROTEIN DISULFIDES S-GLUTATHIONYLATION THIOL PKA DIAMIDE Oxidative Stress 
A molecular switch of ＂Yin and Yang＂： S-glutathionylation of eNOS turns off NO synthesis and turns on superoxide generation
《Acta Pharmacologica Sinica》2011年第4期415-416,共2页Dayue Darrel DUAN Chiu-yin KWAN 
Nitric oxide （NO） is a short-lived free radical produced endogenously in biological tissues by nitric oxide synthases （NOSs）. Three NOS isoforms, namely NOS1 or neuronal NOS （nNOS）, NOS2 or inducible NOS （iNOS...
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