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期刊=Neural Regeneration Research

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Suppressing high mobility group box-1 release alleviates morphine tolerance via the adenosine5'-monophosphate-activated protein kinase/heme oxygenase-1 pathway被引量：1: 《Neural Regeneration Research》2023年第9期2067-2074,共8页Tong-Tong Lin Chun-Yi Jiang Lei Sheng Li Wan Wen Fan Jin-Can Li Xiao-Di Sun Chen-Jie Xu Liang Hu Xue-Feng Wu Yuan Han Wen-Tao Liu Yin-Bing Pan; supported by the National Natural Science Foundation of China;Nos.81971047 (to WTL) and 82073910 (to XFW);the Natural Science Foundation of Jiangsu Province,No.BK20191253 (to XFW);Key R&D Program (Social Development) Project of Jiangsu Province,No.BE2019 732 (to WTL);Jiangsu Province Hospital (the First Affiliated Hospital of Nanjing Medical University) Clinical Capacity Enhancement Project,No.JSPH-511B2018-8 (to YBP)。; Opioids,such as morphine,are the most potent drugs used to treat pain.Long-term use results in high tolerance to morphine.High mobility group box-1(HMGB1) has been shown to participate in neuropathic or inflammatory p...; 关键词：adenosine 5’-monophosphate-activated protein kinase heme oxygenase-1 high mobility group box-1 INTERLEUKIN-1Β MICROGLIA morphine tolerance NEUROINFLAMMATION neuron nuclear factor-κB p65 Toll-like receptor 4

The therapeutic potential of targeting exchange protein directly activated by cyclic adenosine 3',5'-monophosphate(Epac)for central nervous system trauma被引量：3: 《Neural Regeneration Research》2021年第3期460-469,共10页Alba Guijarro-Belmar Dominik Mateusz Domanski Xuenong Bo Derryck Shewan Wenlong Huang; supported by Scottish Rugby Union funding to WH and DS;the NRB PhD scholarship from the International Spinal Rsesarch Trust to AGB;a Hot-Start Scholarship from the University of aberdeen to DD。; Millions of people worldwide are affected by traumatic spinal cord injury,which usually results in permanent sensorimotor disability.Damage to the spinal cord leads to a series of detrimental events including ischaemi...; 关键词：ASTROCYTES axonal regeneration cAMP central nervous system regeneration Epac glial scar microglia NEUROINFLAMMATION neurons spinal cord spinal cord injury traumatic spinal cord injury

Treadmill training improves neurological deficits and suppresses neuronal apoptosis in cerebral ischemic stroke rats被引量：5: 《Neural Regeneration Research》2019年第8期1387-1393,共7页Li-Mei Cao Zhi-Qiang Dong Qiang Li Xu Chen; supported by Clinical Study on Treatment of Cerebral Small Blood Vessel Disease by Integrated Traditional Chinese and Western Medicine,No.ZHYY-ZXYJHZX-201625; RehabilNation training is believed to be beneficial to patients with stroke, but its molecular mechanism is still unclear. Rat models of cerebral ischemic stroke were established by middle cerebral artery occlusion/re...; 关键词：nerve REGENERATION ischemic stroke TREADMILL training neuronal DEFICIT apoptosis cyclic adenosine MONOPHOSPHATE protein kinase A GLYCOGEN synthase kinase-3^ NEUROPROTECTIVE effect neural REGENERATION

How does resveratrol influence the genesis of some neurodegenerative diseases?被引量：1: 《Neural Regeneration Research》2016年第1期86-87,共2页Ester Tellone Antonio Galtieri Annamaria Russo Silvana Ficarra; Advancing age and increased lifespan of human populations worldwide in the mid-nineteenth century,have led to a significant increase in the incidence of neurodegenerative diseases one of the major cause of disability ...; 关键词：resveratrol disability populations genesis superoxide neuronal chromosome prevention incidence monophosphate

Adaptive and regulatory mechanisms in aged rats with postoperative cognitive dysfunction被引量：18: 《Neural Regeneration Research》2014年第5期534-539,共6页Yanlin Bi Shuyun Liu Xinjuan Yu Mingshan Wang Yuelan Wang; Inflammation may play a role in postoperative cognitive dysfunction. 5＇ Adenosine monophos- phate-activated protein kinase, nuclear factor-kappa B, interleukin-1β, and tumor necrosis factor-a are involved in inflamm...; 关键词：nerve regeneration postoperative cognitive dysfunction SPLENECTOMY BRAIN AGING 5＇ad- enosine monophosphate-activated protein kinase nuclear factor-kappa B tumor necrosis factor-α interleukin- 1β neural regeneration

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